Anetoderma
Anetoderma (also known as "Anetoderma maculosa,"[1] "Anetoderma maculosa cutis,"[1] "Atrophia maculosa cutis,"[1] and "Macular atrophy"[2]) is a localized laxity of the skin with herniation or outpouching resulting from abnormal dermal elastic tissue.[2] Anetoderma comes in three types:
- Primary anetoderma
- Jadassohn–Pellizzari anetoderma is a benign condition with focal loss of dermal elastic tissue.[3] Jadassohn-Pellizzari is one of two major classifications of primary anetoderma, the other being Schweninger–Buzzi anetoderma. The difference between the two is that Jadassohn–Pellizzari anetoderma is preceded by inflammatory lesions.[1]
- Schweninger–Buzzi anetoderma is a cutaneous condition characterized by loss of dermal elastic tissue.[1]
- Secondary anetoderma
- Familial anetoderma
See also
References
- ↑ 1.0 1.1 1.2 1.3 1.4 Rapini, Ronald P.; Bolognia, Jean L.; Jorizzo, Joseph L. (2007). Dermatology: 2-Volume Set. St. Louis: Mosby. ISBN 1-4160-2999-0.
- ↑ 2.0 2.1 Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine. (6th ed.). Page 1027. McGraw-Hill. ISBN 0-07-138076-0.
- ↑ Laumann, Anne; Minocha, Julia Sanger; Ho, Stephen C (2009-06-22). "Anetoderma". eMedicine. Web MD. Retrieved 17 December 2009.
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| Epidermal thickening | |
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| Necrobiosis/granuloma | |
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| Dermis/ localized CTD | |
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| Description |
- Anatomy
- Physiology
- Development
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| Disease |
- Infections
- Vesiculobullous
- Dermatitis and eczema
- Papulosquamous
- Urticaria and erythema
- Radiation-related
- Pigmentation
- Mucinoses
- Keratosis, ulcer, atrophy, and necrobiosis
- Vasculitis
- Fat
- Neutrophilic and eosinophilic
- Congenital
- Neoplasms and cancer
- nevi and melanomas
- epidermis
- dermis
- Symptoms and signs
- Terminology
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| Treatment |
- Procedures
- Drugs
- antibiotics
- disinfectants
- emollients and protectives
- itch
- psoriasis
- other
- Wound and ulcer
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