Dyslexia

Dyslexia is a very broad term defining a learning disability that impairs a person's fluency or comprehension accuracy in being able to read,^[1] and which can manifest itself as a difficulty with phonological awareness, phonological decoding, orthographic coding, auditory short-term memory, or rapid naming.^[2][3] Dyslexia is separate and distinct from reading difficulties resulting from other causes, such as a non-neurological deficiency with vision or hearing, or from poor or inadequate reading instruction.^[4][5] It is believed that dyslexia can affect between 5 to 10 percent of a given population although there have been no studies to indicate an accurate percentage.^[6][7][8]

There are three proposed cognitive subtypes of dyslexia: auditory, visual and attentional.^{[7][9][10][11][12][13]} Reading disabilities, or dyslexia, is the most common learning disability, although in research literature it is considered to be a receptive language-based learning disability.^[14]

Accomplished adult dyslexics may be able to read with good comprehension, but they tend to read more slowly than non-dyslexics and may perform more poorly at nonsense word reading (a measure of phonological awareness) and spelling.^[15] Dyslexia is not an intellectual disability, since dyslexia and IQ are not interrelated, as a result of cognition developing independently.^[16]

Classification

Spoken language is a universal form of human communication. The visual notation of written language is not found in all cultures and is relatively new with regards to human evolution.^[17]

There are many definitions of dyslexia but no official consensus has been reached.

The World Federation of Neurology defines dyslexia as "a disorder manifested by difficulty in learning to read despite conventional instruction, adequate intelligence and sociocultural opportunity".^[18]

MedlinePlus and the National Institutes of Health define dyslexia as "a reading disability resulting from the inability to process graphic symbols".^[19]

The National Institute of Neurological Disorders and Stroke gives the following definition for dyslexia:

"Dyslexia is a brain-based type of learning disability that specifically impairs a person's ability to read. These individuals typically read at levels significantly lower than expected despite having normal intelligence. Although the disorder varies from person to person, common characteristics among people with dyslexia are difficulty with spelling, phonological processing (the manipulation of sounds), and/or rapid visual-verbal responding. In adults, dyslexia usually occurs after a brain injury or in the context of dementia. It can also be inherited in some families and so on, and recent studies have identified a number of genes that may predispose an individual to developing dyslexia".^[1]

Other published definitions are purely descriptive or embody causal theories. Varying definitions are used for dyslexia from researchers and organizations around the world; it appears that this disorder encompasses a number of reading skills, deficits and difficulties with a number of causes rather than a single condition.^[20][21]

Castles and Coltheart describe phonological and surface types of developmental dyslexia by analogy to classical subtypes of alexia (acquired dyslexia) which are classified according to the rate of errors in reading non-words.^[22][23] However, the distinction between surface and phonological dyslexia has not replaced the old empirical terminology of dysphonetic versus dyseidetic types of dyslexia.^[21][23][24] The surface/phonological distinction is only descriptive, and devoid of any aetiological assumption as to the underlying brain mechanisms (Galaburda and Cestnick 2003).^[25] Studies have, however, alluded to potential differential underlying brain mechanisms in these populations given performance differences (Cestnick et al.).^[26][27][28] The dysphonetic/dyseidetic distinction refers to two different mechanisms; one that relates to a speech discrimination deficit, and another that relates to a visual perception impairment.

Signs and symptoms

The symptoms of dyslexia vary according to the severity of the disorder as well as the age of the individual.

Preschool-aged children

It is difficult to obtain a certain diagnosis of dyslexia before a child begins school, but many dyslexic individuals have a history of difficulties that began well before kindergarten. Children who exhibit these symptoms early in life have a higher likelihood of being diagnosed as dyslexic than other children. These symptoms include:

• delays in speech^[29]
• slow learning of new words
• difficulty in rhyming words, as in nursery rhymes
• low letter knowledge
• letter reversal or mirror writing^[30][31] (for example, "Я" instead of "R")

Early primary school children

• Difficulty learning the alphabet or letter order
• Difficulty with associating sounds with the letters that represent them (sound-symbol correspondence)
• Difficulty identifying or generating rhyming words, or counting syllables in words^[32] (phonological awareness)
• Difficulty segmenting words into individual sounds, or blending sounds to make words^[33] (phonemic awareness)
• Difficulty with word retrieval or naming problems^[34][35][36]
• Difficulty learning to decode written words
• Difficulty distinguishing between similar sounds in words; mixing up sounds in polysyllabic words (auditory discrimination) (for example, "aminal" for animal, "bisghetti" for spaghetti)

Older primary school children

• Slow or inaccurate reading
• Very poor spelling^[37] which has been called dysorthographia (orthographic coding)
• Difficulty reading out loud, reading words in the wrong order, skipping words and sometimes saying a word similar to another word (auditory processing disorder)
• Difficulty associating individual words with their correct meanings
• Difficulty with time keeping and concept of time when doing a certain task
• Difficulty with organization skills (working memory)
• Children with dyslexia may fail to see (and occasionally to hear) similarities and differences in letters and words, may not recognize the spacing that organizes letters into separate words, and may be unable to sound out the pronunciation of an unfamiliar word (auditory processing disorder)
• Tendencies to omit or add letters or words when writing and reading^[38]

Secondary school children and adults

Some people with dyslexia are able to disguise their weaknesses, even from themselves. Many students reach higher education before they encounter the threshold at which they are no longer able to compensate for their learning weaknesses.

One common misconception about dyslexia is that dyslexic readers write words backwards or move letters around when reading. In fact, this only occurs in a very small population of dyslexic readers. Dyslexic people are better identified by writing that does not seem to match their level of intelligence from prior observations. Additionally, dyslexic people often substitute similar-looking, but unrelated, words in place of the ones intended (what/want, say/saw, help/held, run/fun, fell/fall, to/too, who/how etc.).

Comorbidities

Several learning disabilities often occur with dyslexia, but it is unclear whether these learning disabilities share underlying neurological causes with dyslexia.^[39] These disabilities include, but are not limited to:

• Dysgraphia— a disorder which expresses itself primarily through writing or typing, although in some cases it may also affect eye–hand coordination direction or sequence oriented processes such as tying knots or carrying out a repetitive task. In dyslexia, dysgraphia is often multifactorial, due to impaired letter writing automaticity, finger motor sequencing challenges, organizational and elaborative difficulties, and impaired visual word form which makes it more difficult to retrieve the visual picture of words required for spelling. Dysgraphia is distinct from dyspraxia in that dyspraxia is simply related motor sequence impairment.
• Dyscalculia— a neurological condition characterized by a problem with basic sense of number and quantity and difficult retrieving rote math facts. Often people with this condition can understand very complex mathematical concepts and principles but have difficulty retrieving basic math facts involving addition and subtraction.
• Attention Deficit Disorder — a high degree of co-morbidity has been reported between ADD / ADHD and dyslexia/reading disorders,^[40] although the contributions of dyslexia-related challenges such as auditory verbal working memory to attention issues has not been well established

Cause

Since the symptoms of dyslexia were first identified by Oswald Berkhan in 1881,^[41] and the term 'dyslexia' coined in 1887 by Rudolf Berlin,^[42][43] generations of researchers have been investigating what dyslexia is and trying to identify the biological causes. The theories of the etiology of dyslexia have and are evolving with each new generation of dyslexia researchers, and the more recent theories of dyslexia tend to enhance one or more of the older theories as understanding of the nature of dyslexia evolves. Theories should not be viewed as competing, but as attempting to explain the underlying causes of a similar set of symptoms from a variety of research perspectives and background.^[44][45]

Effect of language orthography

The complexity of a language's orthography or spelling system – formally, its orthographic depth – has a direct impact on how difficult it is to learn to read that language. English has a comparatively deep orthography within the Latin alphabet writing system, with a complex orthographic structure that employs spelling patterns at several levels: principally, letter-sound correspondences, syllables, and morphemes. Other languages, such as Spanish, have alphabetic orthographies that employ only letter-sound correspondences, so-called shallow orthographies. It is relatively easy to learn to read languages like Spanish; it is much more difficult to learn to read languages with more complex orthographies, such as English.^[46] Logographic writing systems, notably Japanese and Chinese characters, have graphemes that aren't linked directly to their pronunciation, which pose a different type of dyslexic difficulty.^{[13][47][48][49]}

From a neurological perspective, different types of writing system, for example alphabetic as compared to logographic writing systems, require different neurological pathways in order to read, write and spell. Because different writing systems require different parts of the brain to process the visual notation of speech, children with reading problems in one language might not have a reading problem in a language with a different orthography. The neurological skills required to perform the tasks of reading, writing, and spelling can vary between different writing systems and as a result different neurological deficits can cause dyslexic problems in relation to different orthographies.^[47][48][49]

Cross-cultural prevalence

Cross-cultural study of the prevalence of dyslexia is difficult as different scholars and different countries often use different criteria to distinguish the cases of dyslexia in the continuum between the able and delayed readers at schools. According to the existing literature, the prevalence of dyslexia can vary widely between cultures. For example, Christall reports differences between 1% and 33%.^[50] Some populations (for example, Japanese and Chinese) may have relatively small number of dyslexic schoolchildren,^[51] and some populations (for example, European, and particularly sub-Saharan African populations) might have higher dyslexia prevalence. According to some researchers, despite the significant differences between the writing systems, Italian, German and English populations suffer similarly from Dyslexia.^[52]

Exacerbating conditions

Dyslexia is attributed to neurological factors that influence the individual's ability to read, write, and spell written language.^[23]

The following conditions may be contributory or overlapping factors, as they can lead to difficulty in reading:

• Aphasia - neurologically based speech disorders, which can cause alexia (acquired dyslexia).
• Attention deficit hyperactivity disorder -^[29][53][54] A disorder that occurs in between 12% and 24% of those with dyslexia.^[8]
• Auditory processing disorder - A condition that affects the ability to process auditory information. Auditory processing disorder is a listening disability.^[55] It can lead to problems with auditory memory and auditory sequencing. Many people with dyslexia have auditory processing problems including history of auditory reversals, and may develop their own logographic cues to compensate for this type of deficit. Auditory processing disorder is recognized as one of the major causes of dyslexia.^{[55][56][57][58]} Some children can acquire auditory processing disorder as a result of experiencing otitis media with effusion (glue ear, sticky ear, grommets) and other severe ear conditions.^[59]
• Developmental dyspraxia -^[29] A neurological condition characterized by a marked difficulty in carrying out routine tasks involving balance, fine-motor control, kinesthetic coordination, difficulty in the use of speech sounds, problems with short term memory and organization are typical of dyspraxics.
• Scotopic sensitivity syndrome, also known as Irlen Syndrome - A term used to describe sensitivity to certain wavelengths of light which interfere with visual processing.^[60][61]
• Specific language impairment (SLI) - A developmental language disorder that can affect both expressive and receptive language. SLI is defined as a "pure" language impairment, meaning that is not related to or caused by other developmental disorders, hearing loss or acquired brain injury. A study by the Universities of Maastricht and Utrecht examined speech perception and speech production in 3-year-old Dutch children at familial risk of developing dyslexia. Their performance in speech sound categorization and their production of words was compared to that of age-matched children with SLI and typically developing controls. The results of the at-risk and SLI-group were highly similar. Analysis of the individual data revealed that both groups contained subgroups with good and poorly performing children. Their impaired expressive phonology seemed to be related to a deficit in speech perception. The findings indicate that both dyslexia and SLI can be explained by a multi-risk model which includes cognitive processes as well as genetic factors.^[29][62]

Experience of speech acquisition delays and speech and language problems can be due to problems processing and decoding auditory input prior to reproducing their own version of speech,^[63][64] and may be observed as stuttering, cluttering or hesitant speech.^[21]

Management

There is no cure for dyslexia, but dyslexic individuals can learn to read and write with appropriate educational support. Early intervention is very helpful.

Especially for undergraduates, some consideration of what 'reading' is and its purpose can be useful. There are techniques (reading the first sentence [and/or last] of each paragraph in a chapter, for example) which can give an overview of content. This can be sufficient for some purposes. Since stress and anxiety are contributors to a dyslexic's weaknesses in absorbing information, removing these can assist in improving understanding. When a dyslexic knows that not every reading experience must be onerous, it greatly helps their mental approach to the task.

The best approaches acknowledge that the objective in helping to improve a dyslexic's 'reading' is not to 'read-like-a-non-dyslexic-does', but to find a way of extracting information from text that works efficiently for someone who processes such information differently from the majority.

For dyslexia intervention with alphabet writing systems the fundamental aim is to increase a child's awareness of correspondences between graphemes and phonemes, and to relate these to reading and spelling. It has been found that training focused towards visual language and orthographic issues yields longer-lasting gains than mere oral phonological training.^[65]

The best form of approach is determined by the underlying neurological cause(s) of the dyslexic symptoms.

Context sensitive spell checkers combined with text-to-speech systems offer forms of assistive technology to dyslexia users, supporting reading and writing.

There is some evidence that the use of specially tailored fonts may provide some measure of assistance for those suffering from dyslexia; however scientific confirmation of this currently appears to be limited to a single master's thesis.^[66][67]

History

• Identified by Oswald Berkhan in 1881,^[68] the term 'dyslexia' was later coined in 1887 by Rudolf Berlin,^[69] an ophthalmologist practising in Stuttgart, Germany,^[70] from the Greek prefix δυσ- (dus-), "hard, bad, difficult"^[71] + λέξις (lexis), "speech, word".^[72][73]
• In 1896, W. Pringle Morgan published a description of a reading-specific learning disorder in the British Medical Journal titled "Congenital Word Blindness".^[74]
• During the 1890s and early 1900s, James Hinshelwood published a series of articles in medical journals describing similar cases of congenital word blindness. In his 1917 book Congenital Word Blindness, Hinshelwood asserted that the primary disability was in visual memory for words and letters, and described symptoms including letter reversals, and difficulties with spelling and reading comprehension.^[75]
• 1925 Samuel T. Orton determined that there was a syndrome unrelated to brain damage that made learning to read difficult. Orton's theory strephosymbolia described individuals with dyslexia having difficulty associating the visual forms of words with their spoken forms.^[76] Orton observed that reading deficits in dyslexia did not seem to stem from strictly visual deficits.^[77] He believed the condition was caused by the failure to establish hemispheric dominance in the brain.^[78] Orton later worked with the psychologist and educator Anna Gillingham to develop an educational intervention that pioneered the use of simultaneous multisensory instruction.^[79]
• In contrast, Dearborn, Gates, Bennet and Blau considered a faulty guidance of the seeing mechanism to be the cause. They sought to discover if a conflict between spontaneous orientation of the scanning action of the eyes from right to left and training aimed at the acquisition of an opposite direction would allow an interpretation of the facts observed in the dyslexic disorder and especially of the ability to mirror-read.
• 1949 Research conducted under G. Mahec show that the phenomenon is clearly linked to the dynamics of sight as it disappears when the space between letters is increased, transforming the reading into spelling. This experience also explains the ability to mirror-read.
• 1968 Makita suggested that dyslexia was mostly absent among Japanese children.^[80] A 2005 study shows that Makita's claim of rarity of incidence of reading disabilities in Japan to be incorrect.^[81]
• In the 1970s a new hypothesis emerged: that dyslexia stems from a deficit in phonological processing or difficulty in recognizing that spoken words are formed by discrete phonemes. Affected individuals have difficulty associating these sounds with the visual letters that make up written words. Key studies suggested the importance of phonological awareness,^[82]
• 1979 Galaburda and Kemper,^[83] and Galaburda et al. 1985,^[84] reported observations from the examination of post autopsy brains of people with dyslexia. Their studies reporting observed anatomical differences in the language center in a dyslexic brain, taken with the similar work of Cohen et al. 1989,^[85] suggested abnormal cortical development, which was presumed to occur before or during the sixth month of foetal brain development.^[23]
• 1993 Castles and Coltheart describe developmental dyslexia as two prevalent and distinct varieties using the subtypes of Alexia, Surface and Phonological Dyslexia.^[22] Manis et al. 1996, concluded that there were probably more than two subtypes of dyslexia, which would be related to multiple underlying deficits.^[86] Cestnick and Coltheart (1999) demonstrated what these underlying deficits are in part, through unveiling different profiles of phonological versus surface dyslexics.^[26] Cestnick and Jerger (2000)^[27] and Cestnick (2001)^[28] further demonstrated distinct processing differences between phonological and surface dyslexics.
• 1994 From post autopsy specimens Galaburda et al., reported: Abnormal auditory processing in people with dyslexia suggests that accompanying anatomical abnormalities might be present in the auditory system. Supported the reported behavioral findings of a left hemisphere-based phonological defect in dyslexic individuals.^[87]
• The development of neuroimaging technologies during the 1980s and 1990s enabled dyslexia research to make significant advances. Positron emission tomography (PET) and functional magnetic resonance imaging (fMRI) studies have revealed the neural signature of adult normal reading (e.g. Fiez and Petersen, 1998;^[88] Turkeltaub et al., 2002^[89] and phonological processing (e.g., Gelfand and Bookheimer, 2003;^[90] Poldrack et al., 1999).^[91] Employing various experimental approaches and paradigms (e.g., the detection or judgment of rhymes, nonword reading, and implicit reading), these studies have localized dysfunctional phonological processing in dyslexia to left-hemisphere perisylvian regions, especially for the alphabetic writing system (Paulesu et al., 2001; for review, see Eden and Zeffiro, 1998,^[92]). However, it has been demonstrated that in nonalphabetic scripts, where reading places less demands on phonemic processing and the integration of visual-orthographic information is crucial, dyslexia is associated with under activity of the left middle frontal gyrus (Siok et al., 2004).^[93]
• 1999 Wydell and Butterworth reported the case study of an English-Japanese bilingual with monolingual dyslexia.^[49] Suggesting that any language where orthography-to-phonology mapping is transparent, or even opaque, or any language whose orthographic unit representing sound is coarse (i.e. at a whole character or word level) should not produce a high incidence of developmental phonological dyslexia, and that orthography can influence dyslexic symptoms.
• 2003 Ziegler and colleagues claimed that the dyslexia suffered by German or Italian dyslexics is very similar to the one suffered by English dyslexics (readers of different—shallow versus deep orthographic systems), supporting the idea that the origin of dyslexia is mostly biological.^[94]
• 2007 Lyytinen et al. Researchers are seeking a link between the neurological and genetic findings, and the reading disorder.^[65]
• 2008 S Heim et al. in a paper titled "Cognitive subtypes of dyslexia" describe how they compared different sub-groups of dyslexics in comparison with a control group. This is one of the first studies not to just compare dyslexics with a non dyslexic control, but to go further and compared the different cognitive sub groups with a non dyslexic control group.^[10]
• 2008 Wai Ting Siok et al. in a paper titled "A structural–functional basis for dyslexia in the cortex of Chinese readers" describe how dyslexia is language dependent, and especially between alphabetic and non-alphabetic writing systems.^[48]
• 2010 KK Chung et al. investigated the "Cognitive profiles of Hong Kong Chinese adolescents with dyslexia".^[13]

Society and culture

Education law

There are many different national legal statutes and different national special education support structures with regard to special education provision which relate to the management of dyslexia.

Film, television, and literature

There have been a number of films, television programs, and works of fiction which focus on the topic of dyslexia.

Research

The majority of currently available dyslexia research relates to the alphabetic writing system, and especially to languages of European origin. However, substantial research is also available regarding dyslexia for speakers of Arabic, Chinese, and Hebrew.^{[25][95][96][97][98]}

Neuroimaging

Modern neuroimaging techniques such as functional magnetic resonance imaging (fMRI) and positron emission tomography (PET) have produced clear evidence of structural differences in the brains of children with reading difficulties. It has been found that people with dyslexia have a deficit in parts of the left hemisphere of the brain involved in reading, which includes the inferior frontal gyrus, inferior parietal lobule, and middle and ventral temporal cortex.^[99]

That dyslexia is neurobiological in origin is supported by what Lyon et al. proclaimed as "overwhelming and converging data from functional brain imaging investigations" (2003, p. 3). The results of these studies suggest that there are observable differences in how the dyslexic brain functions when compared to the brain of a typical reader. Using fMRI, Shaywitz found that good readers show a consistent pattern of strong activation in the back of the brain with weaker activation in the front of the brain during reading tasks. In contrast, the brain activation pattern in dyslexics is the opposite during reading tasks—the frontal part of the brain becomes overactive with weaker activation in the back. Shaywitz points out "It is as if these struggling readers are using the systems in the front of the brain to try to compensate for the disruption in the back of the brain."^[100]

Brain activation studies using PET to study language have produced a breakthrough in understanding of the neural basis of language over the past decade. A neural basis for the visual lexicon and for auditory verbal short term memory components have been proposed,^[101] with some implication that the observed neural manifestation of developmental dyslexia is task-specific (i.e., functional rather than structural).^[102]

A University of Hong Kong study argues that dyslexia affects different structural parts of children's brains depending on the language which the children read.^[48] The study focused on comparing children that were raised reading English and children raised reading Chinese. This is supported in a review by T. Hadzibeganovic et al. (2010).^[103]

A University of Maastricht (Netherlands) study revealed that adult dyslexic readers underactivate superior temporal cortex for the integration of letters and speech sounds.^[104]

Genetic

High genetic concordance found in twin studies suggest a significant genetic influence on reading ability ^{[105] [106]} , although the degree depends on the definition of dyslexia ^[107]. Linkage analysis and genetic association studies (typically quantitative trait locus association studies, which use microarrays to look at single nucleotide polymorphisms of multiple genes at once) have been used to identify candidate genes that may be implicated in dyslexia. ^[108] Several genes have been linked to dyslexia, including DCDC2^[109][110] and KIAA0319^[109][111] on chromosome 6,^[17][112] and DYX1C1 on chromosome 15.^[17][109], ROBO1^[113], DYX3^[114], the language-disorder candidate gene CMIP^[115], and several others. However, these genes account for a small proportion of variance in reading disability, often less than 0.5%.^[116][117] Additionally, the findings are not always replicated. Therefore, no single gene is definitively implicated in dyslexia. A 2007 review reported that no specific cognitive processes are known to be influenced by the proposed genes.^[118]

It likely that multiple genes, as well as the environment, interact to influence reading ability. The Generalist Genes Hypothesis proposes that many of the same genes are implicated within different aspects of a learning disability as well as between different learning disabilities. Indeed, there also appear to be a large genetic influence on other learning abilities, such as language skills. ^[119] The Generalist Genes Hypothesis supports the findings that many learning disabilities are comorbid, such as speech sound disorder, language impairment, and reading disability ^[120]; although this is also influenced by diagnostic overlap.

Many of the genes implicated in dyslexia play a role in general neural development. For example, dyslexia candidate genes DYX1C1, ROBO1 KIAA0319, and DCDC2 appear to be involved in neuronal migration^{[121] [122][123] [124]}. Animal models are especially useful in determining the function of these genes. For example, Gene knockdown in utero of DYX1C1 disrupts hippocampal development and causes impairments in auditory processing and spatial learning in rodents^[125] and mutations in DCDC2 impairs visuo-spatial memory, visual discrimination, and long-term memory in mice^[126]. The role of neuronal migration in dyslexia is reviewed in Galaburda (2005). ^[127]

Gene x Environment

Research has examined gene x environment interactions in reading disability through twin studies, which estimate the proportion of variance associated with environment and the proportion associated with heritability. If the proportion of one increases, the other must decrease, because they sum to 1. Studies examining the influence of environmental factors such as parental education ^[128], and teacher quality ^[129] have determined that genetics influence phenotype much more in supportive environments than less-optimal environments, supporting the bioecological model of gene x environment interactions. However, this does not rule out the possibility that the diathesis-stress model, which proposes that a phenotype depends on a genetic predisposition in combination with a certain environmental stressor, also occurs. An excellent review of the analysis and interpretation of gene x environment interactions, as well as a review of these interactions in reading disability can be found in Pennington et al. 2009. As stated in this reveiw, "it would be a mistake to consider the diathesis-stress and bioecological models simple opposites of each other, because the nature of the underlying process in each is different (Rutter, 2006). In a diathesis-stress interaction found with molecular methods, we assume that both the diathesis and the stress affect the same specific biological substrate and that the two may be jointly necessary for the phenotype to be observed….In contrast, a bioecological interaction found with either molecular or behavioral genetic methods can occur just because a variety of environmental risk factors have been reduced in a favorable environment, and therefore the environment will contribute less to individual differences, and genes will contribute more. Unlike in a diathesis-stress interaction, the environmental factor in a bioecological interaction does not necessarily act on the same biological substrate as the genetic risk factors. Instead, it may just allow those genetic risk factors to account for more of the variance in outcome, because environmental risk factors that affect that outcome have been minimized." ^[130]

As environment plays a large role in learning and memory^[131] , is likely that epigenetic modifications play an important role in reading ability. Animal models and measures of gene expression and methylation in the human periphery are used to study epigenetic processes, both of which have limitations in extrapolating to the human brain.

Controversy

In recent years there has been significant debate on the categorization of dyslexia. In particular, Elliot and Gibbs argue that "attempts to distinguish between categories of 'dyslexia' and 'poor reader' or 'reading disabled' are scientifically unsupportable, arbitrary and thus potentially discriminatory".^[132]

While acknowledging that reading disability is a valid scientific curiosity, and that "seeking greater understanding of the relationship between visual symbols and spoken language is crucial" and that while there was "potential of genetics and neuroscience for guiding assessment and educational practice at some stage in the future", they conclude that "there is a mistaken belief that current knowledge in these fields is sufficient to justify a category of dyslexia as a subset of those who encounter reading difficulties".

See also

• Child development
• History of the alphabet
• Learning theory (education)
• Linguistics
• List of people diagnosed with dyslexia
• Neurolinguistics
• Philosophy of language
• Writing system
• Neurodiversity

References

External links

Research papers, articles and media

• Auditory and Visual Dynamic Processing: Separate Influences in Reading? by Caroline Witton, Joel B. Talcott, Peter C. Hansen, Catherine J. Stoodley & John F. Stein
• Evaluating Alternative Solutions for Dyslexia by Professor Dorothy Bishop

Organizations

• International Dyslexia Association
• Dyslexia Action (UK)
• British Dyslexia Association
• Dyslexia Research Trust