APLP2

Amyloid beta (A4) precursor-like protein 2
Identifiers
Symbols APLP2; APLP-2; APPH; APPL2; CDEBP
External IDs OMIM104776 HomoloGene20396 GeneCards: APLP2 Gene
RNA expression pattern
More reference expression data
Orthologs
Species Human Mouse
Entrez 334 11804
Ensembl ENSG00000084234 ENSMUSG00000031996
UniProt Q06481 n/a
RefSeq (mRNA) NM_001142276.1 n/a
RefSeq (protein) NP_001135748.1 n/a
Location (UCSC) Chr 11:
129.94 – 130.01 Mb
Chr 9:
30.96 – 31.02 Mb
PubMed search [1] [2]

Amyloid beta (A4) precursor-like protein 2, also known as APLP2, is a protein that in humans is encoded by the APLP2 gene.[1][2] APLP2 along with APLP1 are important modulators of glucose and insulin homeostasis.[3]

Contents

Function

APLP2 associates with antigen presentation molecules like MHC class I molecules and regulates their surface expression by enhancing endocytosis.[4][5]

APLP1 and APLP2 double knockout mice display hypoglycemia and hyperinsulinemia indicating that these two proteins are important modulators of glucose and insulin homeostasis.[3]

Interactions

APLP2 has been shown to interact with APBB1.[6][7]

References

  1. ^ "Entrez Gene: APLP2 Amyloid beta (A4) precursor-like protein 2". http://www.ncbi.nlm.nih.gov/sites/entrez?Db=gene&Cmd=ShowDetailView&TermToSearch=334. 
  2. ^ Leach R, Ko M, Krawetz SA (1999). "Assignment of amyloid-precursor-like protein 2 gene (APLP2) to 11q24 by fluorescent in situ hybridization". Cytogenet. Cell Genet. 87 (3–4): 215–6. doi:10.1159/000015472. PMID 10702673. 
  3. ^ a b Needham BE, Wlodek ME, Ciccotosto GD, Fam BC, Masters CL, Proietto J, Andrikopoulos S, Cappai R (June 2008). "Identification of the Alzheimer's disease amyloid precursor protein (APP) and its homologue APLP2 as essential modulators of glucose and insulin homeostasis and growth". J. Pathol. 215 (2): 155–63. doi:10.1002/path.2343. PMID 18393365. 
  4. ^ Tuli A, Sharma M, Naslavsky N, Caplan S, Solheim JC (June 2008). "Specificity of amyloid precursor-like protein 2 interactions with MHC class I molecules". Immunogenetics 60 (6): 303–13. doi:10.1007/s00251-008-0296-0. PMC 2683759. PMID 18452037. http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pmcentrez&artid=2683759. 
  5. ^ Tuli A, Sharma M, McIlhaney MM, Talmadge JE, Naslavsky N, Caplan S, Solheim JC (August 2008). "Amyloid precursor-like protein 2 increases the endocytosis, instability, and turnover of the H2-K(d) MHC class I molecule". J. Immunol. 181 (3): 1978–87. PMC 2607064. PMID 18641335. http://www.jimmunol.org/cgi/content/abstract/181/3/1978. 
  6. ^ Guénette SY, Chen J, Jondro PD, Tanzi RE (October 1996). "Association of a novel human FE65-like protein with the cytoplasmic domain of the beta-amyloid precursor protein". Proc. Natl. Acad. Sci. U.S.A. 93 (20): 10832–7. doi:10.1073/pnas.93.20.10832. PMC 38241. PMID 8855266. http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pmcentrez&artid=38241. 
  7. ^ Tanahashi H, Tabira T (February 1999). "Molecular cloning of human Fe65L2 and its interaction with the Alzheimer's beta-amyloid precursor protein". Neurosci. Lett. 261 (3): 143–6. doi:10.1016/S0304-3940(98)00995-1. PMID 10081969. 

Further reading