ALDH7A1

Aldehyde dehydrogenase 7 family, member A1

PDB rendering based on 2j6l.
Identifiers
Symbols ALDH7A1; ATQ1; EPD; FLJ11738; FLJ92814; PDE
External IDs OMIM107323 MGI108186 HomoloGene913 GeneCards: ALDH7A1 Gene
EC number 1.2.1.31
Orthologs
Species Human Mouse
Entrez 501 110695
Ensembl ENSG00000164904 ENSMUSG00000053644
UniProt P49419 Q9DBF1
RefSeq (mRNA) NM_001182.4 NM_138600
RefSeq (protein) NP_001173.2 NP_001120810
Location (UCSC) Chr 5:
125.88 – 125.93 Mb
Chr 18:
56.69 – 56.73 Mb
PubMed search [1] [2]

Aldehyde dehydrogenase 7 family, member A1 also known as ALDH7A1 or antiquitin is an enzyme that in humans is encoded by the ALDH7A1 gene.[1]

Contents

Function

Antiquitin is a member of subfamily 7 in the aldehyde dehydrogenase gene family. These enzymes are thought to play a major role in the detoxification of aldehydes generated by alcohol metabolism and lipid peroxidation. This particular member has homology to a previously described protein from the green garden pea, the 26g pea turgor protein.[2]

Antiquitin functions as an aldehyde dehydrogenase in the pipecolic acid pathway of lysine catabolism.[3]

Clinical significance

Mutations in this gene cause pyridoxine-dependent epilepsy, which involves a combination of various seizure types and is responsive to immediate administration of pyridoxine hydrochloride.[3]

References

  1. ^ Skvorak AB, Robertson NG, Yin Y, Weremowicz S, Her H, Bieber FR, Beisel KW, Lynch ED, Beier DR, Morton CC (December 1997). "An ancient conserved gene expressed in the human inner ear: identification, expression analysis, and chromosomal mapping of human and mouse antiquitin (ATQ1)". Genomics 46 (2): 191–9. doi:10.1006/geno.1997.5026. PMID 9417906. 
  2. ^ "Entrez Gene: ALDH7A1". http://www.ncbi.nlm.nih.gov/sites/entrez?Db=gene&Cmd=ShowDetailView&TermToSearch=501. 
  3. ^ a b Mills PB, Struys E, Jakobs C, Plecko B, Baxter P, Baumgartner M, Willemsen MA, Omran H, Tacke U, Uhlenberg B, Weschke B, Clayton PT (March 2006). "Mutations in antiquitin in individuals with pyridoxine-dependent seizures". Nature Medicine 12 (3): 307–9. doi:10.1038/nm1366. PMID 16491085. 

External links

Further reading

This article incorporates text from the United States National Library of Medicine, which is in the public domain.