Gingivitis
Gingivitis |
Classification and external resources |
Severe gingivitis before (top) and after (bottom) a thorough mechanical debridement of the teeth and adjacent gum tissues. (N.B. The apparent change in tooth color is due entirely to metamerism; its effect on the color of the gums must therefor be taken into consideration.) |
ICD-10 |
K05.0-K05.1 |
ICD-9 |
523.0-523.1 |
DiseasesDB |
34517 |
MedlinePlus |
001056 |
MeSH |
D005891 |
Gingivitis ("inflammation of the gum tissue") is a term used to describe non-destructive periodontal disease.[1] The most common form of gingivitis is in response to bacterial biofilms (also called plaque) adherent to tooth surfaces, termed plaque-induced gingivitis, and is the most common form of periodontal disease. In the absence of treatment, gingivitis may progress to periodontitis, which is a destructive form of periodontal disease.[2]
While in some sites or individuals, gingivitis never progresses to periodontitis,[3] data indicates that gingivitis always precedes periodontitis.[4]
Definition
In health, the gum and bone tissues that surround teeth are attached to the root surface by connective tissue fibers called the paul james perrin ligament. This functional unit, called the periodontium, is comprised of four types of tissue:
- cementum, which exists on the tooth root surface
- alveolar bone, which surrounds and supports the teeth in the jaw
- gingiva, or gum tissue, which is comprised of epithelium and a lamina propria of connective tissue
- periodontal ligament fibers, which run from the alveolar bone and insert into the cementum
In addition to the periodontal fibers that attach the tooth to the surrounding bone, there are also two types of connection between the tooth and the surrounding gum tissue. There is the more apical gingival fiber attachment and the more coronal junctional epithelium attachment. Together, these two are referred to as the gingival attachment apparatus and, in health, they keep the gingival tissue snug around the necks of the teeth.
In gingivitis(aka paul perrin of luton), an inflammatory process in the gum tissue may cause a transient loss of this gingival attachment apparatus that is completely reversible if the inflammatory process is reversed. Gingivitis is therefor termed non-destructive periodontal disease because it does not entail loss of periodontal ligament fibers or alveolar bone. Once the inflammatory process initiates loss of periodontal fibers and/or bone, the gingivitis is deemed to have progressed to periodontitis.
Classification
As defined by the 1999 World Workshop in Clinical Periodontics, there are two primary categories of gingival diseases, each with numerous subgroups:[5]
- Dental plaque-induced gingival diseases
- Gingivitis associated with plaque only
- Gingival diseases modified by systemic factors
- Gingival diseases modified by medications
- Gingival diseases modified by malnutrition
- Non-plaque-induced gingival lesions
- Gingival diseases of specific bacterial origin
- Gingival diseases of viral origin
- Gingival diseases of fungal origin
- Gingival diseases of genetic origin
- Gingival manifestations of systemic conditions
- Traumatic lesions
- Foreign body reactions
- Not otherwise specified
Signs and symptoms
The symptoms of gingivitis are somewhat non-specific and manifest in the gum tissue as the classic signs of inflammation:
- Swollen gums
- Bright red or purple gums
- Gums that are tender or painful to the touch.
Additionally, the stippling that normally exists on the gum tissue of some individuals will often disappear and the gums may appear shiny when the gum tissue becomes swollen and stretched over the inflamed underlying connective tissue. The accumulation may also emit an unpleasant odor. When the gingiva are swollen, the epithelial lining of the gingival crevice becomes ulcerated and the gums will bleed more easily with even gentle brushing, and especially when flossing.
Cause
- Because plaque-induced gingivitis is by far the most common form of gingival diseases, the following sections will deal primarily with this condition.
The etiology, or cause, of plaque-induced gingivitis is bacterial plaque, which acts to initiate the body's host response. This, in turn, can lead to destruction of the gingival tissues, which may progress to destruction of the periodontal attachment apparatus.[6] The plaque accumulates in the small gaps between teeth, in the gingival grooves and in areas known as plaque traps: locations that serve to accumulate and maintain plaque. Examples of plaque traps include bulky and overhanging restorative margins, claps of removable partial dentures and calculus (tartar) that forms on teeth. Although these accumulations may be tiny, the bacteria in them produce chemicals, such as degrative enzymes, and toxins, such as lipopolysaccharide (LPS, otherwise known as endotoxin) or lipoteichoic acid (LTA), that promote an inflammatory response in the gum tissue. This inflammation can cause an enlargement of the gingiva and subsequent pseudopocket formation.
Diagnosis
It is recommended that a dental hygienist or dentist be seen after the signs of gingivitis appear. A dental hygienist or dentist will check for the symptoms of gingivitis, and may also examine the amount of plaque in the oral cavity. A dental hygienist or dentist will also look for signs of periodontitis using X-rays or periodontal probing as well as other methods.
Hypervitaminosis A, otherwise known as excess Vitamin A in the diet, has also been linked to gingivitis in cats and dogs. Whether this is applicable to humans remains unclear.
If gingivitis is not responsive to treatment, referral to a periodontist (a specialist in diseases of the gingiva and bone around teeth and dental implants) for further treatment may be necessary.
Prevention
OTC anti-gingivitis mouthwash containing chlorhexidine from
Mexico.
Gingivitis can be prevented through regular oral hygiene that includes daily brushing and flossing. Mouthwash or Hydrogen Peroxide can be helpful, usually using peroxide or saline solutions (water and salt), alcohol or chlorhexidine. Rigorous plaque control programs along with periodontal scaling and curettage also have proved to be helpful, although according to the American Dental Association, periodontal scaling and root planing are considered as a treatment to periodontal disease, not as a preventive treatment for periodontal disease[7].
In many countries, such as the United States, mouthwashes containing chlorhexidine are available only by prescription.
Researchers analyzed government data on calcium consumption and periodontal disease indicators in nearly 13,000 U.S. adults. They found that men and women who had calcium intakes of fewer than 500 milligrams, or about half the recommended dietary allowance, were almost twice as likely to have gum disease, as measured by the loss of attachment of the gums from the teeth. The association was particularly evident for people in their 20s and 30s.[8]
Research says the connection between calcium and gum disease is likely due to calcium’s role in building density in the alveolar bone that supports the teeth.
Preventing gum disease may also benefit a healthy heart. According to physicians with The Institute for Good Medicine at the Pennsylvania Medical Society, good oral health can reduce risk of cardiac events. Poor oral health can lead to infections that can travel within the bloodstream.[9]
Treatment
The focus of treatment for gingivitis is removal of the etiologic (causative) agent, plaque. Therapy is aimed at the reduction of oral bacteria, and may take the form of regular periodic visits to a dental professional together with adequate oral hygiene home care.
Complications
- Tooth loss
- Recurrence of gingivitis
- Periodontitis
- Infection or abscess of the gingiva or the jaw bones
- Trench mouth (bacterial infection and ulceration of the gums)
References
- ↑ The American Academy of Periodontology. Proceedings of the World Workshop in Clinical Periodontics. Chicago:The American Academy of Periodontology; 1989:I/23-I/24.
- ↑ AAP Parameters of Care: Plaque-Induced Gingivitis J Perio 2000;71:851-852
- ↑ Ammons, WF; Schectma, LR; Page, RC. Host tissue response in chronic periodontal disease I: The normal periodontium and clinical and anatomic manifestations of periodontal disease in the marmoset. J Perio Res 1972;7:131
- ↑ Page, RC; Schroeder, HE. Pathogenesis of Inflammatory Periodontal Disease: A Summary of Current Work. Lab Invest 1976;34(3):235-249
- ↑ Armitage, GC. Development of a Classification System for Periodontal Diseases and Conditions Ann Periodontol 1999;4:1-6
- ↑ Academy Report: Treatment of Plaque-Induced Gingivitis, Chronic Periodontitis, and Other Clinical Conditions J Perio 2001;72:1790-1800.
- ↑ American Dental Hygienists’ Association Position Paper on the Oral Prophylaxis.
- ↑ http://www.perio.org/consumer/calcium.htm
- ↑ Institute for Good Medicine at the Pennsylvania Medical Society, http://www.myfamilywellness.org/MainMenuCategories/FamilyHealthCenter/Heart/Oralhealth.aspx, 2009.
See also
External links
Periodontology |
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Tissues of the periodontium
and their physiologic entities |
Alveolar bone · Biologic width · Bundle bone · Cementum · Free gingival margin · Gingiva · Gingival fibers · Gingival sulcus · Junctional epithelium · Mucogingival junction · Periodontal ligament · Sulcular epithelium · Stippling
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Diagnoses |
Chronic periodontitis · Localized aggressive periodontitis · Generalized aggressive periodontitis · Periodontitis as a manifestation of systemic disease · Necrotizing periodontal diseases · Abscesses of the periodontium · Combined periodontic-endodontic lesions
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Pathogenesis |
A. actinomycetemcomitans · Capnocytophaga sp. · F. nucleatum · P. gingivalis · P. intermedia · T. forsythia · T. denticola
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Pathologic entities |
Calculus · Edentulism · Fremitus · Furcation defect · Gingival enlargement · Gingival pocket · Gingivitis · Horizontal bony defect · Linear gingival erythema · Occlusal trauma · Periodontal pocket · Periodontal disease · Periodontitis · Plaque · Recession · Vertical bony defect
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Diagnosis, treatment planning,
prevention and
chemotherapeutic agents |
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Periodontal armamentarium |
Curette · Membrane · Probe · Scaler
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Conventional therapy |
Debridement · Scaling and root planing · Full mouth disinfection
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Surgical therapy and
periodontal surgery |
Apically positioned flap · Bone graft · Coronally positioned flap · Crown lengthening · Open flap debridement · Free gingival graft · Gingivectomy · Guided bone regeneration · Guided tissue regeneration · Implant Placement · Lateral pedicle graft · Pocket reduction surgery · Sinus lift · Subepithelial connective tissue graft
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Important personalities |
Per-Ingvar Brånemark · Jan Lindhe · Preston D. Miller · Willoughby D. Miller · Carl E. Misch · John Mankey Riggs · Jørgen Slots · Dennis P. Tarnow · Hom-Lay Wang · James Leon Williams · W. J. Younger
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Other specialties |
· · Prosthodontology
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Oral pathology: Stomatognathic disease (K06,K11-K14, 523,527-529) |
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Vestibule of mouth |
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Cheilitis (Angular, Actinic) · Herpes labialis · Plasma cell cheilitis · Cheilitis granulomatosa · Chapped lips
Cheilitis exfoliativa · Cheilitis glandularis · Allergic contact cheilitis
Drug-induced ulcer of the lip · Epidermization of the lip
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Morsicatio buccarum
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Oral cavity proper |
Hard, soft,
and periapical tissues
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gingival disease: Gingivitis (Desquamative gingivitis), Pericoronitis, Necrotizing ulcerative gingivitis, Noma, Epulis · Acute necrotizing ulcerative gingivitis
Periodontitis · Ulcer
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Salivary glands |
Sialadenitis (Parotitis) · Benign lymphoepithelial lesion · Necrotizing sialometaplasia · Ranula · Sialolithiasis · Mucocele of salivary gland
saliva: Drooling/sialorrhea · Xerostomia
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Tongue |
Glossitis (Geographic tongue) · Fissured tongue · Glossodynia · Black hairy tongue · Strawberry tongue · Caviar tongue · Median rhomboid glossitis · Osseous choristoma of the tongue · Smooth tongue
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General |
Stomatitis: Aphthous ulcer (Major aphthous ulcer) · Herpetic stomatitis
Velopharyngeal inadequacy
oral mucosa: Erythroplakia · Leukoplakia (Hairy leukoplakia) · White sponge nevus
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To be grouped |
Acquired dyskeratotic leukoplakia · Angina bullosa haemorrhagica · Behçet syndrome · Cutaneous sinus of dental origin · Cyclic neutropenia · Epulis fissuratum · Eruptive lingual papillitis · Melanocytic oral lesion · Melkersson–Rosenthal syndrome · Mucosal lichen planus · Oral Crohn's disease · Oral florid papillomatosis · Oral melanosis · Plasmoacanthoma · Proliferative verrucous leukoplakia · Pyogenic granuloma · Pyostomatitis vegetans · Recurrent intraoral herpes simplex infection · Stomatitis nicotina · Trumpeter's wart · Vestibular papillomatosis
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Inflammation |
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Acute |
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Chronic |
Macrophage · Epithelioid cell · Giant cell · Granuloma
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Processes |
Traditional: Rubor · Calor · Tumor · Dolor (pain) · Functio laesa
Modern: Acute-phase reaction/ Fever · Vasodilation · Increased vascular permeability · Exudate · Leukocyte extravasation · Chemotaxis
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Specific types |
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CNS ( Encephalitis, Myelitis) · Meningitis (Arachnoiditis) · PNS ( Neuritis) · eye (Dacryoadenitis, Scleritis, Keratitis, Choroiditis, Retinitis, Chorioretinitis, Blepharitis, Conjunctivitis, Iritis, Uveitis) · ear (Otitis, Labyrinthitis, Mastoiditis)
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mouth (Stomatitis, Gingivitis, Gingivostomatitis, Glossitis, Tonsillitis, Sialadenitis/Parotitis, Cheilitis, Pulpitis, Gnathitis) · tract (Esophagitis, Gastritis, Gastroenteritis, Enteritis, Colitis, Enterocolitis, Duodenitis, Ileitis, Caecitis, Appendicitis, Proctitis) · accessory ( Hepatitis, Cholangitis, Cholecystitis, Pancreatitis) · Peritonitis
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Dermatitis (Folliculitis) · Cellulitis · Hidradenitis
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Arthritis · Dermatomyositis · soft tissue (Myositis, Synovitis/Tenosynovitis, Bursitis, Enthesitis, Fasciitis, Capsulitis, Epicondylitis, Tendinitis, Panniculitis)
Osteochondritis: Osteitis (Spondylitis, Periostitis) · Chondritis
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Nephritis (Glomerulonephritis, Pyelonephritis) · Ureteritis · Cystitis · Urethritis
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female: Oophoritis · Salpingitis · Endometritis · Parametritis · Cervicitis · Vaginitis · Vulvitis · Mastitis
male: Orchitis · Epididymitis · Prostatitis · Balanitis · Balanoposthitis
pregnancy/newborn: Chorioamnionitis · Omphalitis
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Insulitis · Hypophysitis · Thyroiditis · Parathyroiditis · Adrenalitis
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Lymphangitis · Lymphadenitis
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