TP73L
From Wikipedia, the free encyclopedia
Tumor protein p73-like, also known as TP73L, is a human gene. It is associated with ectrodactyly-ectodermal dysplasia-cleft syndrome and Hay-Wells syndrome.
[edit] Further reading
- Little NA, Jochemsen AG (2002). "p63.". Int. J. Biochem. Cell Biol. 34 (1): 6–9. PMID 11733180.
- van Bokhoven H, McKeon F (2002). "Mutations in the p53 homolog p63: allele-specific developmental syndromes in humans.". Trends in molecular medicine 8 (3): 133–9. PMID 11879774.
- van Bokhoven H, Brunner HG (2002). "Splitting p63.". Am. J. Hum. Genet. 71 (1): 1–13. PMID 12037717.
- Brunner HG, Hamel BC, Bokhoven Hv H (2003). "P63 gene mutations and human developmental syndromes.". Am. J. Med. Genet. 112 (3): 284–90. doi: . PMID 12357472.
- Jacobs WB, Walsh GS, Miller FD (2005). "Neuronal survival and p73/p63/p53: a family affair.". The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry 10 (5): 443–55. doi: . PMID 15359011.
- Zusman I (2005). "The soluble p51 protein in cancer diagnosis, prevention and therapy.". In Vivo 19 (3): 591–8. PMID 15875781.
- Morasso MI, Radoja N (2005). "Dlx genes, p63, and ectodermal dysplasias.". Birth Defects Res. C Embryo Today 75 (3): 163–71. doi: . PMID 16187309.
- Barbieri CE, Pietenpol JA (2006). "p63 and epithelial biology.". Exp. Cell Res. 312 (6): 695–706. doi: . PMID 16406339.