Talk:Periosteal reaction

From Wikipedia, the free encyclopedia

The content of this article has been derived in whole or part from http://uwmsk.org/residentprojects/ and/or http://uwmsk.org/moodle/course/view.php?id=44. Permission has been received from the copyright holder to license this material under the GNU Free Documentation License, and evidence of this has been lodged with the Wikimedia PR department. This template should not be used to make a claim of permission or ownership, but only when a clear permission statement has been forwarded to permissions at wikimedia dot org.

Discussion and verification of permission is archived at Talk:Jumper's Knee. Melchoir 23:42, 17 April 2006 (UTC)

[edit] Text from page

I hate to do it, but the main page is very complicated and the multiple editors don't seem to be de-complicating things. I've pasted this massive chunk of text and pictures to be gradually reintroduced to make my life slightly easier. Bleah. The tone is very out of keeping with WP:MEDMOS, it looks very much like a doctor wrote it or pasted it from a textbook. Way too high a level for a general reader. WLU (talk) 21:47, 19 December 2007 (UTC)

Did some re-editing, still can't re-integrate much into the text.

---

Radiologic findings

Primary hypertrophic osteoarthropathy

Hypertrophic osteopathy is a clinical syndrome demonstrating clubbing, painful swollen joints, enlargement of the extremities due to osseus proliferation, and thickening of the skin over the face and scalp. An individual may have all or any combination of the above features. Primary hypertrophic osteoarthropathy is the hereditary form and accounts for only 3-5% of all hypertrophic osteoarthropathy. It has autosomal dominant inheritance with variable expression. Radiographs demonstrate irregular or fluffy periosteal reaction that most often involves the long tubular bones of the extremities - radius, ulna, tibia, and fibula. The metacarpals, phalanges, and pelvis can also be involved. Resorption of the phalangeal tufts has also been described. In the primary form, the periostitis is usually not painful.

Secondary hypertrophic osteoarthropathy

Secondary hypertrophic osteoathropathy is the acquired form and has a high association with diseases of the chest, occurring in up to 5% of individuals with with bronchogenic carcinoma and up to 50% of patients with mesothelioma. Cardiac and gastrointestinal diseases, although frequent, also are associated with development of the disease. The mechanism by which these diverse conditions induce disease is unknown. In the cases of abscesses and lung cancer, it has been noted the patient's symptoms resolve within a day following thoracotomy. Clinically patients with the secondary form of the disease tend to have pain that is often worse at night. From a radiographic standpoint the periosteal reaction can be single- or multilayered-linear or irregular, such as with the primary form. Overall the distribution is similar to the primary form with a predilection for tubular long bones of the extremities.

Periostitis

The frequency of periostitis increases with the duration and severity of chronic venous stasis; it is reported in 10-60% of patients. The radiographic findings tend to be distributed in the lower extremities of these patients from femur to the metatarsals.

Caffrey's disease is an uncommon form of hyperostosis and periosteal reaction that occurs in newborns. There is usually very dense periosteal new bone formation. It can occur at any site, but the mandible, clavicle, and scapula, and ribs are most commonly involved.

Primary hypertrophic osteoarthropathy. The distal radius and ulna show diffuse new bone formation that also affects the metacarpus	left|Secondary hypertrophic osteoarthropathy attributed to fungal infection.	Chest radiograph demonstrating fungal infection	Secondary hypertrophic osteoarthropathy in a patient with lung neoplasm.
Chest radiograph demonstrating lung neoplasm.	Periosteal new bone formation in a patient with chronic venous stasis.