KCNJ13

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Potassium inwardly-rectifying channel, subfamily J, member 13
Identifiers
Symbol(s) KCNJ13; KIR1.4; KIR7.1; MGC33328
External IDs OMIM: 603208 HomoloGene: 55638
RNA expression pattern

More reference expression data
Orthologs
Human Mouse
Entrez 3769 n/a
Ensembl ENSG00000115474 n/a
Uniprot O60928 n/a
Refseq NM_002242 (mRNA)
NP_002233 (protein)		 n/a (mRNA)
n/a (protein)
Location Chr 2: 233.34 - 233.35 Mb n/a
Pubmed search [1] n/a

Potassium inwardly-rectifying channel, subfamily J, member 13 (KCNJ13), also known as KIR7.1, is a human gene.[1]


Contents

[edit] See also

[edit] References

  1. ^ Entrez Gene: KCNJ13 potassium inwardly-rectifying channel, subfamily J, member 13.

[edit] Further reading

  • Kubo Y, Adelman JP, Clapham DE, et al. (2006). "International Union of Pharmacology. LIV. Nomenclature and molecular relationships of inwardly rectifying potassium channels.". Pharmacol. Rev. 57 (4): 509-26. doi:10.1124/pr.57.4.11. PMID 16382105. 
  • Krapivinsky G, Medina I, Eng L, et al. (1998). "A novel inward rectifier K+ channel with unique pore properties.". Neuron 20 (5): 995-1005. PMID 9620703. 
  • Partiseti M, Collura V, Agnel M, et al. (1998). "Cloning and characterization of a novel human inwardly rectifying potassium channel predominantly expressed in small intestine.". FEBS Lett. 434 (1-2): 171-6. PMID 9738472. 
  • Döring F, Derst C, Wischmeyer E, et al. (1998). "The epithelial inward rectifier channel Kir7.1 displays unusual K+ permeation properties.". J. Neurosci. 18 (21): 8625-36. PMID 9786970. 
  • Derst C, Döring F, Preisig-Müller R, et al. (1999). "Partial gene structure and assignment to chromosome 2q37 of the human inwardly rectifying K+ channel (Kir7.1) gene (KCNJ13).". Genomics 54 (3): 560-3. doi:10.1006/geno.1998.5598. PMID 9878260. 
  • Nakamura N, Suzuki Y, Sakuta H, et al. (1999). "Inwardly rectifying K+ channel Kir7.1 is highly expressed in thyroid follicular cells, intestinal epithelial cells and choroid plexus epithelial cells: implication for a functional coupling with Na+,K+-ATPase.". Biochem. J. 342 ( Pt 2): 329-36. PMID 10455019. 
  • Strausberg RL, Feingold EA, Grouse LH, et al. (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences.". Proc. Natl. Acad. Sci. U.S.A. 99 (26): 16899-903. doi:10.1073/pnas.242603899. PMID 12477932. 
  • Gerhard DS, Wagner L, Feingold EA, et al. (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC).". Genome Res. 14 (10B): 2121-7. doi:10.1101/gr.2596504. PMID 15489334. 
  • Kimura K, Wakamatsu A, Suzuki Y, et al. (2006). "Diversification of transcriptional modulation: large-scale identification and characterization of putative alternative promoters of human genes.". Genome Res. 16 (1): 55-65. doi:10.1101/gr.4039406. PMID 16344560. 

[edit] External links

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Membrane transport protein: ion channels
Ca2+: Calcium channel
Na+: Sodium channel
Navα (1.1, 1.2, 1.3, 1.4, 1.5, 1.6, 1.7, 1.9, 7A) • Navβ (1, 2, 3, 4) • Epithelial sodium channel
K+: Potassium channel
Cl-: Chloride channel
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Cations: TRP
TRPA (1) • TRPC (1, 2, 3, 4, 4AP, 5, 6, 7) • TRPM (1, 2, 3, 4, 5, 6, 7, 8) • TRPML (Mucolipin-1) • TRPP (1, 2) • TRPV (1, 2, 3, 4, 5, 6)
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