Talk:Intracranial pressure
From Wikipedia, the free encyclopedia
 |
This article is within the scope of WikiProject Neurology. Please visit the project page for details or ask questions at the talk page. |
| B | This page has been rated as B-Class on the quality assessment scale |
| High | This article has been rated as High-importance on the importance assessment scale |
 |
This article is within the scope of WikiProject Medicine. Please visit the project page for details or ask questions at the doctor's mess. |
| B | This page has been rated as B-Class on the quality assessment scale |
| Mid | This article has been rated as Mid-importance on the importance assessment scale |
This page needs work! Will try in coming months to sit down and write a full paper.~ Neuroscientist July 4, 2005 13:29 (UTC)
[edit] Linking Ischemia
There are references ot Ischemia in this article, which can be found in another article in the wikipedia here: http://en.wikipedia.org/wiki/Ischemia
Just thought they should be linked. I'd do it if I knew how.
Cheers —The preceding unsigned comment was added by 219.95.59.9 (talk • contribs) .
[edit] This page has so many falsities it isn't funny.
The whole premise that if your ICP goes beyond 40mm/hg you will suffer irrepairble damage is false. I have managed many TBI patients who have had sustained ICP's in the high 30's and 40's for days with decent recovery of functionl. Some but not all go on to work and function in society several are in college or gainfully returned to work. The premise of only taking the ICP in to account with out weighing other factors is indicative of a complete patient mismanagement and delenquncy of the medical team doing so. Cou can not monitor ICP alone with out taking in to account the perfusion pressures(MAP-ICP=CPP) and end capillary brain tissue oxygenation(pbtO2)in the penumbra of the injury. Since 2002 any reputable medical center that has managed patients with elevated ICP's has done so knowing that they must maintain adequate cerebral perfusion. When the brain loses compliance the perfusion thresholds increase not entirely unlike someone with noncompliant lungs needing positive pressure ventilation to recruit alveoli and exchange gases. Guided brain tissue oxygenation and cerebral microdialysis are vanguard in assuring that these types of patients get the very best care. It's the brain, stupid!
[edit] Question on another page
There's a question at Talk:Orthostatic_hypotension#Question which might be appropriate (or interesting) to address in this page. The question is about the nonpathologic perception of increased pressure in the head from reclining with the head below the heart. I think that, for the average reader, an explanation of how pressure is normally regulated, and what the body does to keep a handstand from killing you, might be interesting. WhatamIdoing (talk) 18:31, 9 February 2008 (UTC)
