Talk:Premature ejaculation

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I reverted the insertion of yoinked copyrighted text, and added an external link to the apparent source. -- Infrogmation 22:24, 20 Dec 2004 (UTC)

I removed more copyrighted text ("Inability to constantly control the ejaculatory reflex...") from the Armenian Medical Network. -- Kslays 18:48, 2 August 2006 (UTC)

The article makes it sound as if SSRIs (selective serotonin reuptake inhibitors) reduce prematue ejaculation through reducing anxiety. I am not at all an expert, but my impression was that SSRIs just make orgasm more difficult as a "side effect", i.e. not by way of reducing anxiety. Can anyone who knows the facts confirm? -- Bayle Shanks

SSRI's will reduce anxiety, that is for sure. They will also make it harder for both man and women to get aroused, and, once aroused, they will make it harder, or even impossible in some cases, to achieve an orgasm. So there is probably some of both of these effects.

[edit] heterocentric?

I think this article should be changed, at the moment it implies that only straight men have ejaculatory problems. this could just be remedied by simply changing the language from female to just partner.

WP:BB. Or: {{sofixit}}. JFW | T@lk 22:31, 16 February 2006 (UTC)

Okay, changed! just wanted to see if anyone had some objections first :D

Explain this to me: Women need vaginal stimulation to climax which can take time, whereas with two men having sex the "catcher" is not going to climax by having a penis in his bum. So trying to include gay sex in here is moot. I'm changing it back because I don't think it's normal for a man having a penis in his bum to orgasm due to having a penis in his bum.

On top of that, I think peoople get the picture without having to list every type of sex out there. If you include sexual positions that deviate from functional sex that can produce offspring (i.e. inserting the penis into a non-functional cavity such as an anus) then you also have to list mouth, boobs, hand, chicken, cow, sheep, etc... People get the picture when you list the normal method and if they want to translate that to other types of sex then that is their business. JettaMann 15:54, 27 July 2006 (UTC)

JettaMann I agree with your point, bravo for putting the PC monster back in its cage. But in fact it is possible for a male "receiver" to orgasm (by internal stimulation of the prostate.) Even some women can climax anally, go figure the human body is an amazing contraption. You may want to read up on a few homosexual articles, if you can stomach it :) --Jquarry 12:19, 15 August 2006 (UTC)

Um, women don't necessarily need vaginal stimulation to climax; we need clitoral stimulation. 70% of women do not reach orgasm from plain old vaginal intercourse, so Master and Johnsons' definition (man climaxes before woman more than 50% of the time) does not make a whole lot of sense to me. Rosemary Amey 23:14, 17 November 2006 (UTC)

[edit] SSRIs

SSRIs do alleviate PE, but this is not to do with increasing the level of seratonin in the synapse. If that were the case, more straightforward seratonin boosters (such as l-tryptophan or 5-htp) would also alleviate PE, which they do not - at least, not to anywhere near the same extent. A better explanation is the fact that SSRIs block re-uptake of seratonin - the male orgasm involves a huge re-uptake of seratonin (this is what creates the "whooshing" sensation as a man goes past the point of no return), and by blocking this, SSRIs prevent uncontrolled ejaculation. This also explains why many men taking SSRIs have to concentrate quite hard in order to orgasm at all (and thus override the drug effect), and why some men on SSRIs cannot orgasm at all. This would also explain why St John's Wort (which does block re-uptake) is more effective for PE than 5-htp / l-tryptophan.

I'd add this myself, but I don't have a citation to hand, so it could be considered original research - the role of seratonin re-uptake in male orgasm, the effect of SSRIs on seratonin re-uptake, and the efficacy of SSRIs in treating PE are all common knowledge, but I've yet to read a detailed, peer-reviewed scientific article connecting the three. Then again, I've certainly never read one which makes the case that SSRIs achieve this effect by increasing seratonin in the synapse! Perhaps someone could dig up citations for the three points made above, and present the idea that way. It's certainly a more scientifically credible idea than the one currently in the article MrBronson 19:42, 24 October 2006 (UTC)